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However, the physiological and pathophysiological roles of GSH remain largely unexplored in vivo, substantially limiting our ability to therapeutically target GSH in disease contexts

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582 Preclinical evidence suggests that NAFLD/NASH-related HCC is characterized by reduced CD4+ T cell activity within tumors, loss of tumor surveillance function by CD8+PD-1+T cells, and pro-tumorigenic functions of NKT cells and Th17 cells