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While both treatments promoted mesenchymal thinning via cell death, endotoxin uniquely suppressed the expression of elastic fibers and induced chronic inflammatory stress, possibly disrupting lung maturation in the long term (308)

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Metabolic dysfunction-associated steatohepatitis (MASH) is a more dangerous form of MASLD, characterized by histological findings of inflammation in the liver parenchyma, hepatocyte ballooning with increased hepatocyte necrosis, and an elevated risk of fibrosis progression [7]