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Since SAMe is a necessary cofactor for catechol-o-methyltransferase (COMT) metabolism of catecholamines [53,54], and since FAD acts to cofactor the monoamine oxidase [MAO] enzyme [72], interruption of SAMe synthesis together with low FAD availability, compromises catecholamine metabolism at three levels of MAO, COMT and conversion of noradrenaline [NA] to adrenaline [AD] [55], leading to conserved, unmetabolized dopamine (DA) and noradrenaline (NA)

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Among the 44 clinical and metabolic features, glutamic acid was the strongest predictor according to SHAP analysis