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Revered as a “gatekeeper” in virtue of its function and localization, ABCG2 stands guard, thwarting the passage of endogenous or exogenous toxins and foreign substances across biological barricades into delicate tissues [49]

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This microenvironment-driven conversion is supported by the transcriptional upregulation of key stemness regulators such as OCT4, NANOG, and SOX2 and is further reinforced by epigenetic modifications such as promoter methylation or histone acetylation, which stabilize the reprogrammed state