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Some studies [55, 56, 57] showed that each catalase tetramer has four tightly bound NADPH molecules that appear to be important only to protect the enzyme against inactivation by its own substrate (H 2 O 2 ), and are not essential for its catalytic activity

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CAE provides proof-of-principle that an endogenous disruption of myelin can trigger MS-like brain lesions in an immune-predisposed host

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