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κB-Ras proteins function in both Ral and NF-κB signaling pathways (Oeckinghaus et al, 2014), and attribution of the observed in vivo phenotype of κB-Ras–deficient mice to deregulation of Ral signaling was circumstantial

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Blood collection, demographic, and clinical data from study cohorts were approved by Stanford University’s IRB (IRB-40146, IRB-64344)