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Oral administration of Enterococcus faecalis FK-23 suppresses Th17 cell development and attenuates allergic airway responses in mice

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The intestinal leakage hypothesis suggests that the reduction of cardiac output and the increase of systemic congestion will cause intestinal mucosal ischemia and edema, which will further lead to the increase of intestinal permeability to proinflammatory mediators (Zhang et al., 2015