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Studies have shown that complement components C3 and C5 are maintained at high levels in blood-derived neutrophils and monocytes from both humans and mice

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boulardii) has demonstrated mycotoxin binding effects, 16 as well as the ability to reverse mycotoxin-induced injury by attenuating the “p38 mitogen-activated protein kinase (MAPK)” signaling pathway to modulate expression of inflammatory cytokines and apoptotic genes