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Consequent mechanistic studies revealed that L-theanine, a glutamate analogue, competes with glutamate to bind the glutamate receptor, resulting in suppression of chemotherapy efflux (extracellular transport), increasing their concentration (27)

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First, the upregulation of Psme3, hnRNP K, and H2BC3 suggests that the ATM/P53-dependent DNA damage checkpoint is fully activated and that G 2 /M cell cycle arrest is maintained for several hours until repair is effected and cells reenter the cell cycle 26