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In view of (a) the potentially similar metabolic effects of G6PD and GR deficiency, (b) the well documented protection from severe falciparum malaria afforded by G6PD deficiency [14], [19] and (c) the fact that P

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It arrests the cell cycle (G2/M phase) by CDK2/4 downregulation and triggers ROS production, leading to activation of apoptotic cell signaling in glioblastoma cells [225–227]