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Studies indicate that QACR can hinder the proliferation of HCC cells by targeting the c-Jun N-terminal kinase signaling pathway ( In vitro studies, QACR has been shown to inhibit the growth of MHCC97-L and SK-Hep-1 cells, induce cell apoptosis, reduce levels of PCNA, Bcl-2, and CD31 proteins, enhance expression of apoptosis-related proteins like caspase 3, caspase 8, caspase 9, and DFF40, and decrease HUVEC cell angiogenesis